Try to cut out the food that you think does this the most. For me a lot of foods give me GERD. Like spice, onions, dark chocolate etc. But tea was the worst. Once I stopped drinking tea, things got much better. You must have one food like that that is doing this to you.

(http://www.autoadmit.com/thread.php?thread_id=3222939&forum_id=2ﾃ｢竄ï½���„1¤77PHPSESSID=3d8e678aec504db068978830e992758d#30474505)

I made a change shortly after that and it went away, no major issues.

(http://www.autoadmit.com/thread.php?thread_id=3222939&forum_id=2ﾃ｢竄ï½���„1¤77PHPSESSID=3d8e678aec504db068978830e992758d#30474687)

i didn't take any meds

(http://www.autoadmit.com/thread.php?thread_id=3222939&forum_id=2ﾃ｢竄ï½���„1¤77PHPSESSID=3d8e678aec504db068978830e992758d#30474709)

42 tablets of 20 mg prilosec = $23 on target's website right now.

90 tablets of 40 mg omeprazole = $10

(http://www.autoadmit.com/thread.php?thread_id=3222939&forum_id=2ﾃ｢竄ï½���„1¤77PHPSESSID=3d8e678aec504db068978830e992758d#30474836)

http://www.medscape.com/viewarticle/859438 (feb 26, 2016)

This study was based on the follow-up of a smaller cohort study that had been reported in 2015, suggesting a similar association with a hazard ratio (HR) of about 1.4 for PPI use and dementia.[5] In the present study of more than 200,000 patients, approximately 29,000 received a diagnosis of dementia in the 11 years of follow-up. Dementia was categorized just as dementia, and only 2.7% of patients actually had Alzheimer disease.

The scientific hypothesis was that PPIs may change the development of beta-amyloid plaques, and that there is some potential, at least shown in mice, that the PPIs may alter the beta-secretase or gamma-secretase that lays down these plaques in the brain.[6] As you may or may not know, there is some hypothesis and demonstration that these amyloid plaques are seen in the Alzheimer variant of dementia. Now recalling how dementia was categorized in this study and that only 2.7% of patients had Alzheimer disease, it's important to put this percentage into perspective, given this hypothesis that PPIs may change the development of amyloid plaques.

There was a nominal difference between men and women, with an HR of 1.52 for men and an HR of 1.42 for women for the association between PPI use and dementia.

The authors adjusted for some variables, but certainly not all confounders. They adjusted for age, gender, polypharmacy, stroke history, depression, ischemic heart disease, and diabetes.

They didn't adjust for some very important risk factors for dementia, including alcohol use, family history of dementia, and hypertension. These are well-known risk factors for dementia, and for some reason, these weren't included in the analysis. Thus, we're missing a tremendous amount of potential risk factors that might account for the imbalance of incident dementia between PPI users and nonusers.

We need to recognize that "dementia" was not well qualified. We have no way to know whether it was true dementia. Was it just cognitive impairment? There clearly are psychomotor and cognitive assessments that can differentiate cognitive impairment without dementia. We don't have any of that information.

As in a number of these recent pharmacovigilant studies, we've seen a nominal odds ratio association suggesting associated with a medication; in this case, it's a PPI. Interestingly, the same challenge was made with histamine-2 (H2) receptor antagonists. A study reported that H2 antagonist users had about 2.4 greater odds of cognitive impairment compared with nonusers.[7] This one is 1.4 times. In another study, H2 antagonist use was not associated with all-cause dementia or Alzheimer disease.[8]

There was also no risk-profile adjustment for dose. If you were going to report that this was a dose/response effect, you'd like to see some effect of higher doses or over longer periods of time.

In my mind, there are a lot of potential biases owing to the lack of statistical adjustment for key risk factors for dementia. We also don't have any knowledge about the qualitative assessments for the diagnosis of dementia in these patients, but we do know that there are mechanistic ways to do this using validated instruments, none of which were used in this particular study.

There are a lot of issues with these data.

*Some of these pharmacovigilance studies with PPIs have created a lot of undue angst. When carefully analyzed, they really are not anything more than a little bit of noise right now.*

In my mind (no pun intended, and I do take a PPI), I'm very confident that right now you don't need to change therapies on the basis of concern about dementia.

(http://www.autoadmit.com/thread.php?thread_id=3222939&forum_id=2ﾃ｢竄ï½���„1¤77PHPSESSID=3d8e678aec504db068978830e992758d#30474919)

there was a recent medical study that claimed proton-pump inhibitors, over a multi-decade daily course of treatment, slightly increase chance of kidney trouble -- so i asked him about it.

he said not to worry about it; single studies are always being disproven, the jury's still out, and even if it turns out to be true, it's a slight risk after taking it for a very long time -- meaning there's a high likelihood a better treatment comes along before the long-term side effects manifest.

GI doc's been taking 60mg omeprazole daily for over three decades and has never seen any kidney problems in himself or anyone he's treated.

he's undoubtedly full of shit. but before i started taking it, i'd regularly choke up stomach acid while sleeping; hasn't happened again since.

(http://www.autoadmit.com/thread.php?thread_id=3222939&forum_id=2ﾃ｢竄ï½���„1¤77PHPSESSID=3d8e678aec504db068978830e992758d#30474872)

http://www.medscape.com/viewarticle/859438 (feb 26, 2016)

This study was based on the follow-up of a smaller cohort study that had been reported in 2015, suggesting a similar association with a hazard ratio (HR) of about 1.4 for PPI use and dementia.[5] In the present study of more than 200,000 patients, approximately 29,000 received a diagnosis of dementia in the 11 years of follow-up. Dementia was categorized just as dementia, and only 2.7% of patients actually had Alzheimer disease.

The scientific hypothesis was that PPIs may change the development of beta-amyloid plaques, and that there is some potential, at least shown in mice, that the PPIs may alter the beta-secretase or gamma-secretase that lays down these plaques in the brain.[6] As you may or may not know, there is some hypothesis and demonstration that these amyloid plaques are seen in the Alzheimer variant of dementia. Now recalling how dementia was categorized in this study and that only 2.7% of patients had Alzheimer disease, it's important to put this percentage into perspective, given this hypothesis that PPIs may change the development of amyloid plaques.

There was a nominal difference between men and women, with an HR of 1.52 for men and an HR of 1.42 for women for the association between PPI use and dementia.

The authors adjusted for some variables, but certainly not all confounders. They adjusted for age, gender, polypharmacy, stroke history, depression, ischemic heart disease, and diabetes.

They didn't adjust for some very important risk factors for dementia, including alcohol use, family history of dementia, and hypertension. These are well-known risk factors for dementia, and for some reason, these weren't included in the analysis. Thus, we're missing a tremendous amount of potential risk factors that might account for the imbalance of incident dementia between PPI users and nonusers.

We need to recognize that "dementia" was not well qualified. We have no way to know whether it was true dementia. Was it just cognitive impairment? There clearly are psychomotor and cognitive assessments that can differentiate cognitive impairment without dementia. We don't have any of that information.

As in a number of these recent pharmacovigilant studies, we've seen a nominal odds ratio association suggesting associated with a medication; in this case, it's a PPI. Interestingly, the same challenge was made with histamine-2 (H2) receptor antagonists. A study reported that H2 antagonist users had about 2.4 greater odds of cognitive impairment compared with nonusers.[7] This one is 1.4 times. In another study, H2 antagonist use was not associated with all-cause dementia or Alzheimer disease.[8]

There was also no risk-profile adjustment for dose. If you were going to report that this was a dose/response effect, you'd like to see some effect of higher doses or over longer periods of time.

In my mind, there are a lot of potential biases owing to the lack of statistical adjustment for key risk factors for dementia. We also don't have any knowledge about the qualitative assessments for the diagnosis of dementia in these patients, but we do know that there are mechanistic ways to do this using validated instruments, none of which were used in this particular study.

There are a lot of issues with these data.

*Some of these pharmacovigilance studies with PPIs have created a lot of undue angst. When carefully analyzed, they really are not anything more than a little bit of noise right now.*

In my mind (no pun intended, and I do take a PPI), I'm very confident that right now you don't need to change therapies on the basis of concern about dementia.

(http://www.autoadmit.com/thread.php?thread_id=3222939&forum_id=2ﾃ｢竄ï½���„1¤77PHPSESSID=3d8e678aec504db068978830e992758d#30474927)

i would avoid for dinner - fatty foods, spicy foods, dairy

(http://www.autoadmit.com/thread.php?thread_id=3222939&forum_id=2ﾃ｢竄ï½���„1¤77PHPSESSID=3d8e678aec504db068978830e992758d#30474694)

it's not so bad you'll get used to eating more bland food. I still eat plenty of meat just not all the other unnecessary shit that often goes with it

(http://www.autoadmit.com/thread.php?thread_id=3222939&forum_id=2ﾃ｢竄ï½���„1¤77PHPSESSID=3d8e678aec504db068978830e992758d#30474723)

(http://www.autoadmit.com/thread.php?thread_id=3222939&forum_id=2ﾃ｢竄ï½���„1¤77PHPSESSID=3d8e678aec504db068978830e992758d#30474751)

nothing to see here folks.

your going to be ok '"""".

(http://www.autoadmit.com/thread.php?thread_id=3222939&forum_id=2ﾃ｢竄ï½���„1¤77PHPSESSID=3d8e678aec504db068978830e992758d#30474809)

Also consider a prescription for rifaximin. This is used to treat small intestinal bacterial overgrowth disorder, a common cause of digestive trouble.

Do not take any probiotics. Lots of people complain about that shit causing heartburn.

Also consider getting thyroid and liver function checked.

A tablespoon of honey can sooth the stomach, too.

(http://www.autoadmit.com/thread.php?thread_id=3222939&forum_id=2ﾃ｢竄ï½���„1¤77PHPSESSID=3d8e678aec504db068978830e992758d#30474829)

Problems with your liver enzymes as well as fatty liver disorder will also affect digestion quality.

One crucial thing to keep in mind is that too LITTLE hydrochloric acid is as bad, actually worse, than too much.

Doctors are SPS. If you need a med they won't give you, you can try ordering from a Mexican pharmacy online. (At your own risk, I am not a doctor, etc.)

(http://www.autoadmit.com/thread.php?thread_id=3222939&forum_id=2ﾃ｢竄ï½���„1¤77PHPSESSID=3d8e678aec504db068978830e992758d#30474857)

prety sure i had this as a cause a while back

(http://www.autoadmit.com/thread.php?thread_id=3222939&forum_id=2ﾃ｢竄ï½���„1¤77PHPSESSID=3d8e678aec504db068978830e992758d#30474965)

Vomiting excess stomach acid in my sleep: age 25

(http://www.autoadmit.com/thread.php?thread_id=3222939&forum_id=2ﾃ｢竄ï½���„1¤77PHPSESSID=3d8e678aec504db068978830e992758d#30476926)